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A mutant gene can provide a second skeleton

Fibrodysplasia ossificans progressiva (FOP) begins early in childhood and has no cure. It transforms muscles, ligaments and tendons into ribbons, sheaths and plates of bone. It covers and locks the joints that makes movement impossible. US researchers recognized a transformed gene after the research of more than 15 years. It turns muscle and other connective tissues into bone to provide its victims, a second skeleton.

This mutant FOP gene is applicable in the formation of bone and conditions which effects the formation of the skeleton. The findings can develop a drug to treat the rare bone disorder. The drug will be even more helpful for sports injuries.

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